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As a result, it's conceivable the JIP1/3???ASK1???MKK4 signaling module may perhaps function a conduit to activate JNK when endosomal trafficking is disrupted and glucose levels tumble over the early phases of methuosis. Our acquiring that MKK4 was robustly activated because of the methuosis-inducing IPPs, MOMIPP and 2q, and less so from the non-cy